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What is causing my progressive muscle weakness and how can it be treated?
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Nervous System Disorders
Question #30707
19 hours ago
43

What is causing my progressive muscle weakness and how can it be treated? - #30707

Client_3bd2ca

Female, 25, since years progressive muscle weakness. September 2021: First symptoms of muscle fatigue under load, such as brushing my hair or walking fast. There was no clinical weakness yet. 2022: Became completely unable to do sit-ups or push-ups, despite being able to manage 10 to 15 push-ups before. Early 2023: Lost the ability to stand up from a squat without using my hands. By August 2023, standing up from a chair without using my arms became impossible. December 2023: Sudden onset of extreme heaviness starting in the neck muscles and spreading to my abdominal muscles and arms within minutes. Even holding a phone or a blanket felt impossibly heavy. It slowly settled that day but left a permanent decline: my muscles now fatigue much faster, including the jaw, and I can no longer push myself up from seats with poor armrests. April 2024: Going down stairs became limited as my right thigh could no longer cushion the impact after a few steps. February 2025: Following a one-week hospital stay in neurology, my right thigh completely lost the ability to climb even a single step, causing me to fall. My left leg can no longer climb higher steps either. Shortly after this, my right thigh swelled up by an extra 7 centimeters in circumference. May 2025; muscle biopsy showed neurogenic atrophy and a mild myopathy. Summer 2025: The swelling turned out to be a separate condition. After several examinations, a tumor biopsy, and surgery, it was diagnosed as a desmoid tumor, a very rare, benign but locally aggressive tumor. It was completely removed surgically in July, but the removal brought no functional improvement to my leg. September 2025: Following local anesthesia at the dentist, my primary upper lip failed permanently. The sensation returned fully, but the lip no longer lifts when I smile or open my mouth. Two months later, involuntary compensation developed, meaning other facial muscles like the nose elevator fire automatically when I try to smile. The neurologist noted this as a failed reinnervation. Late 2025 into 2026: When sitting or standing with a leaning upper body, other muscles (legs) overcompensate and fatigue within minutes. In moving vehicles, I have to hold on tightly, or my torso will completely tilt to the side when going around curves. I can also no longer pull in my stomach to button my jeans, despite having a stable weight. Currently, my jaw, neck, arms, legs, abdomen, and parts of my face are affected. I do not experience pain, numbness, or any other sensory symptoms. Once I experience a loss of function, it never comes back, despite continuous physiotherapy. Right side is more effected from the weakness even though both sides are effected. Doctors have tried trials of 50mg prednisolone, huperzine A, and D-ribose, but nothing helped. Since local neurologists are overwhelmed by the complexity, I would really appreciate any advice how to continue further, also with the findings of the muscle biopsy showed neurogenic atrophy.

When did you first notice the muscle weakness?:

- More than 2 years ago

Have you experienced any other symptoms besides muscle weakness?:

- No other symptoms

What treatments have you already tried?:

- Physical therapy

How has your muscle strength changed over time?:

- Worsened gradually

Have you had any recent imaging or tests done?:

- MRI

Do you have a family history of similar muscle or neurological conditions?:

- Not sure

How would you describe your overall energy levels?:

- Low most of the time
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Doctors' responses

Dr. Shayeque Reza
I completed my medical degree in 2023, but honestly, my journey in healthcare started way before that. Since 2018, I’ve been actively involved in clinical practice—getting hands-on exposure across multiple departments like ENT, pediatrics, dermatology, ophthalmology, medicine, and emergency care. One of the most intense and defining phases of my training was working at a District Government Hospital for a full year during the COVID pandemic. It was chaotic, unpredictable, and exhausting—but it also grounded me in real-world medicine like no textbook ever could. Over time, I’ve worked in both OPD and IPD setups, handling everything from mild viral fevers to more stubborn, long-term conditions. These day-to-day experiences really built my base and taught me how to stay calm when things get hectic—and how to adjust fast when plans don’t go as expected. What I’ve learned most is that care isn't only about writing the right medicine. It’s about being fully there, listening properly, and making sure the person feels seen—not just treated. Alongside clinical work, I’ve also been exposed to preventive health, health education, and community outreach. These areas really matter to me because I believe real impact begins outside the hospital, with awareness and early intervention. My approach is always centered around clarity, empathy, and clinical logic—I like to make sure every patient knows exactly what’s going on and why we’re doing what we’re doing. I’ve always felt a pull towards general medicine and internal care, and honestly, I’m still learning every single day—each patient brings a new lesson. Medicine never really sits still, it keeps shifting, and I try to shift with it. Not just in terms of what I know, but also in how I listen and respond. For me, it’s always been about giving real care. Genuine, respectful, and the kind that actually helps a person heal—inside and out.
5 hours ago
5

This is a complex and concerning progressive neuromuscular disorder rather than simple deconditioning or fatigue. The key features are: gradual progression over several years, predominantly proximal weakness (difficulty with squats, stairs, rising from chairs, lifting arms), involvement of the neck, jaw, abdominal muscles, facial muscles, and limbs, greater involvement on the right side, absence of sensory symptoms, and permanent loss of function once weakness develops. The muscle biopsy showing both neurogenic atrophy and mild myopathy is particularly important because it suggests that the process may involve both the motor nerves and the muscle itself, rather than a purely muscular disease. The lack of improvement with prednisolone also makes common inflammatory myopathies less likely, although it does not completely exclude them.

Given the history, further evaluation at a specialized neuromuscular center would be highly appropriate if it has not already been done.Conditions that specialists may consider include hereditary motor neuron disorders, distal or limb-girdle muscular dystrophies, facioscapulohumeral muscular dystrophy (FSHD), inclusion body myopathy variants, mitochondrial disorders, congenital myopathies presenting later in life, or rare genetic neuromuscular syndromes. The facial involvement and asymmetric progression are particularly noteworthy. If not already performed, investigations that may be valuable include comprehensive neuromuscular genetic testing (or whole-exome/genome sequencing), detailed EMG and nerve conduction studies, CK levels, repeat expert review of the muscle biopsy, and review of muscle MRI patterns. The persistent upper lip weakness after dental anesthesia may represent a nerve injury, but the neurologist’s observation of failed reinnervation raises the possibility that an underlying neuromuscular disorder is impairing recovery. Overall, this history is most consistent with a progressive neuromuscular disease requiring tertiary-center evaluation, and obtaining consultation at a dedicated neuromuscular clinic—preferably one with expertise in rare genetic and motor neuron disorders—would be the most important next step. The combination of progressive weakness, facial involvement, neurogenic atrophy on biopsy, and lack of sensory symptoms warrants continued investigation rather than attributing the symptoms to a single muscle injury or the previously removed desmoid tumor.

2035 answered questions
59% best answers

2 replies
Client_3bd2ca
Client
4 hours ago

Thank you for your response! My CK have always been consistently normal. The biopsy did not show any ragged red fibers, but few targetoid fibers were detected.

Dr. Shayeque Reza
I completed my medical degree in 2023, but honestly, my journey in healthcare started way before that. Since 2018, I’ve been actively involved in clinical practice—getting hands-on exposure across multiple departments like ENT, pediatrics, dermatology, ophthalmology, medicine, and emergency care. One of the most intense and defining phases of my training was working at a District Government Hospital for a full year during the COVID pandemic. It was chaotic, unpredictable, and exhausting—but it also grounded me in real-world medicine like no textbook ever could. Over time, I’ve worked in both OPD and IPD setups, handling everything from mild viral fevers to more stubborn, long-term conditions. These day-to-day experiences really built my base and taught me how to stay calm when things get hectic—and how to adjust fast when plans don’t go as expected. What I’ve learned most is that care isn't only about writing the right medicine. It’s about being fully there, listening properly, and making sure the person feels seen—not just treated. Alongside clinical work, I’ve also been exposed to preventive health, health education, and community outreach. These areas really matter to me because I believe real impact begins outside the hospital, with awareness and early intervention. My approach is always centered around clarity, empathy, and clinical logic—I like to make sure every patient knows exactly what’s going on and why we’re doing what we’re doing. I’ve always felt a pull towards general medicine and internal care, and honestly, I’m still learning every single day—each patient brings a new lesson. Medicine never really sits still, it keeps shifting, and I try to shift with it. Not just in terms of what I know, but also in how I listen and respond. For me, it’s always been about giving real care. Genuine, respectful, and the kind that actually helps a person heal—inside and out.
4 hours ago
5

Ok. Do give your valuable review.

2035 answered questions
59% best answers
Dr. Arsha K Isac
I am a general dentist with 3+ years of working in real-world setups, and lemme say—every single patient teaches me something diff. It’s not just teeth honestly, it’s people… and how they feel walking into the chair. I try really hard to not make it just a “procedure thing.” I explain stuff in plain words—no confusing dental jargon, just straight talk—coz I feel like when ppl *get* what's going on, they feel safer n that makes all the difference. Worked with all ages—like, little kids who need that gentle nudge about brushing, to older folks who come in with long histories and sometimes just need someone to really sit n listen. It’s weirdly rewarding to see someone walk out lighter, not just 'coz their toothache's gone but coz they felt seen during the whole thing. A lot of ppl come in scared or just unsure, and I honestly take that seriously. I keep the vibe calm. Try to read their mood, don’t rush. I always tell myself—every smile’s got a story, even the broken ones. My thing is: comfort first, then precision. I want the outcome to last, not just look good for a week. Not tryna claim perfection or magic solutions—just consistent, clear, hands-on care where patients feel heard. I think dentistry should *fit* the person, not push them into a box. That's kinda been my philosophy from day one. And yeah, maybe sometimes I overexplain or spend a bit too long checking alignment again but hey, if it means someone eats pain-free or finally smiles wide in pics again? Worth it. Every time.
5 hours ago
5

Hello

The pattern you describe is concerning for a progressive neuromuscular disorder, and the muscle biopsy finding of neurogenic atrophy with mild myopathy is a particularly important clue. Unfortunately, it is not possible to determine the exact diagnosis from the information provided, but several possibilities deserve consideration.

Features that stand out include:

* Slowly progressive weakness over several years * Predominantly proximal weakness (difficulty rising from a squat, chair, climbing stairs, lifting arms) * Facial involvement (persistent upper lip weakness) * Neck, trunk, abdominal, jaw, arm, and leg involvement * No sensory symptoms * Permanent loss of function once weakness develops * Biopsy showing neurogenic atrophy * Lack of response to prednisone

This combination raises concern for disorders affecting the motor neurons, peripheral nerves, neuromuscular junction, or certain genetic muscle diseases. Possibilities that would warrant further evaluation include:

* Adult-onset spinal muscular atrophy (SMA) * Hereditary motor neuropathies * Facioscapulohumeral muscular dystrophy (FSHD) * Limb-girdle muscular dystrophies * Inclusion body myopathy variants (less typical at your age) * Rare genetic neuromuscular syndromes * Less commonly, atypical motor neuron diseases

The fact that prednisone, huperzine A, and D-ribose were ineffective makes an inflammatory muscle disease or classic myasthenia gravis less likely, though not completely excluded.

At this stage, the most valuable next steps would be:

1. Comprehensive EMG and nerve conduction studies, preferably at a specialized neuromuscular center. 2. Genetic testing, ideally a broad neuromuscular gene panel or whole-exome/genome sequencing if not already performed. 3. Review of the muscle biopsy by a neuromuscular pathology expert if this has not been done. 4. Measurement of CK (creatine kinase), respiratory muscle function, and cardiac evaluation if not already completed. 5. Assessment at a tertiary neuromuscular referral center with expertise in rare neuromuscular diseases.

The facial weakness following dental anesthesia is particularly unusual. The return of sensation but persistent inability to elevate the lip suggests either nerve injury with incomplete recovery or an underlying vulnerability of the motor system. The neurologist’s observation of failed reinnervation makes this an important clue rather than something that should automatically be attributed solely to the dental procedure.

Because your weakness is progressive and affecting multiple muscle groups, I would encourage seeking evaluation at a major neuromuscular center rather than continuing isolated local consultations. The biopsy result showing neurogenic atrophy indicates that further investigation of the motor neuron and peripheral motor nerve systems is especially important.

Feel free to talk Take care

1893 answered questions
56% best answers

1 replies
Client_3bd2ca
Client
4 hours ago

Thank you for your response! My CK have always been consistently normal. The biopsy did not show any ragged-red fibers nor was there any histological evidence of myositis. However few targetoid fibers were detectable, alongside the findings of neurogenic atrophy. I have been tested multiple times for both myasthenia gravis and Lambert-Eaton myasthenic syndrome antibodies and the results were consistently negative.

Dr. Prasannajeet Singh Shekhawat
I am a 2023 batch passout and working as a general physician right now, based in Hanumangarh, Rajasthan. Still kinda new in the bigger picture maybe, but honestly—every single day in this line teaches you more than textbooks ever could. I’ve had the chance to work under some pretty respected doctors during and after my graduation, not just for the clinical part but also to see how they handle people, real people, in pain, in panic, and sometimes just confused about their own health. General medicine covers a lot, right? Like from the smallest complaints to those random, vague symptoms that no one really understands at first—those are kinda my zone now. I don’t really rush to label things, I try to spend time actually listening. Feels weird to say it but ya, I do take that part seriously. Some patients just need someone to hear the whole story instead of jumping to prescription pads after 30 seconds. Right now, my practice includes everything from managing common infections, blood pressure issues, sugar problems to more layered cases where symptoms overlap and you gotta just... piece things together. It's not glamorous all the time, but it's real. I’ve handled a bunch of seasonal disease waves too, like dengue surges and viral fevers that hit rural belts hard—Hanumangarh doesn’t get much spotlight but there’s plenty happening out here. Also, I do rely on basics—thorough history, solid clinical exam and yeah when needed, investigations. But not over-prescribing things just cz they’re there. One thing I picked up from the senior consultants I worked with—they used to say “don’t chase labs, chase the patient’s story”... stuck with me till now. Anyway, still learning every single day tbh. But I like that. Keeps me grounded and kind of obsessed with trying to get better.
3 hours ago
5

Hello It sounds like you’re dealing with a complex and challenging situation, especially with the neurogenic atrophy findings from your muscle biopsy. I can understand how frustrating it must be to experience weakness and loss of function without relief from treatments. Here’s a friendly approach to consider as you navigate this:

### Next Steps to Consider

1. Seek a Specialist: - If local neurologists are overwhelmed, consider seeking a referral to a neuromuscular specialist or a neurorehabilitation center. They often have more experience with complex cases and can provide tailored treatment plans.

2. Comprehensive Evaluation: - A thorough evaluation by a specialist may include advanced imaging (like MRI) and additional tests to assess nerve function and muscle health. This can help pinpoint the underlying cause of your symptoms.

3. Physical Therapy: - Continue with physical therapy, but ensure it’s tailored to your specific needs. A physical therapist with experience in neuromuscular conditions can help design a program that focuses on maintaining mobility and strength without exacerbating weakness.

4. Occupational Therapy: - An occupational therapist can assist with daily activities and suggest adaptive strategies or tools to help you manage your symptoms better.

5. Nutritional Support: - Consider consulting a nutritionist who specializes in neuromuscular disorders. Proper nutrition can play a role in muscle health and overall well-being.

6. Explore Alternative Therapies: - Some patients find relief through complementary therapies like acupuncture, massage, or yoga. While these should not replace conventional treatment, they may help improve quality of life.

7. Support Groups: - Connecting with others who have similar conditions can provide emotional support and practical advice. Look for local or online support groups for individuals with neuromuscular disorders.

8. Regular Follow-Ups: - Keep regular follow-up appointments with your healthcare team to monitor your condition and adjust treatment plans as necessary.

### Summary Navigating a complex condition like yours can be daunting, but seeking specialized care and a comprehensive approach can make a difference. Don’t hesitate to advocate for yourself and explore all available options.

Thank you

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